New Theory Explores Role of Carrier Pigeons, Mustard Gas, and Milk in the 1918 Pandemic
Press release published 07 Oct by BEVERAGE MARKETING USA, INC.
Carrier Pigeons and Mustard Gas: The Unseen Connection
Davis's paper, titled *The Most Likely Cause of the Spanish Flu Was Carrier Pigeons Used During the War*, suggests that pigeons used for wartime communication may have harbored a form of bird flu that mutated into the Spanish Flu. This transformation, according to Davis, was triggered by the use of mustard gas in the trenches, which severely weakened the birds' lungs. This, he argues, allowed the virus to thrive in the birds' respiratory systems, facilitating its transmission to soldiers on both the German and Allied sides.
"The birds’ exposure to mustard gas compromised their respiratory health, making them vulnerable to infection. Once the virus took hold, these pigeons became vectors, transmitting the flu through the trenches," said Davis. "The close quarters of the trenches only accelerated the spread among soldiers."
Military Men and Flu Susceptibility: The Milk Hypothesis
Davis also explores the demographics of those most affected by the Spanish Flu, noting that young, military-aged men—many of whom did not consume milk—were disproportionately impacted. His theory posits that those who regularly drank milk, such as women, children, and the elderly, had a natural immunity to the virus due to the protective properties of lactic acid found in milk.
"Milk drinkers seem to have fared better against the Spanish Flu," Davis explains. "The lactic acid and mucus produced by milk consumption likely created a protective barrier in the lungs, preventing the virus from taking hold. It’s no coincidence that teetotalers—those who abstained from alcohol—were also less affected by the flu. They, too, were more likely to consume milk, which may have contributed to their immunity."
Flu Immunity and the Role of Milk
Davis draws on scientific studies that highlight the antiviral properties of milk, particularly breast milk and bovine milk. According to Davis, the compounds in milk, such as lactoferrin and chondroitin sulfate, may have helped reduce the risk of viral infections by inhibiting the binding of viruses to host cells.
"Milk isn't a classical antiviral, but its components can create a protective layer in the mouth and throat that may temporarily wash away viruses," Davis said. "In the case of the Spanish Flu, this mucus-like coating, rich in lactic acid, could have been the difference between life and death for many."
Spanish Flu: A Deadly Pandemic
The Spanish Flu of 1918 killed an estimated 50 million people worldwide, including approximately 43,000 U.S. soldiers. While children and women appeared to experience milder symptoms, young men in the trenches were among the hardest hit. Davis’s hypothesis aims to shed new light on why this may have been the case, particularly in relation to the soldiers’ lifestyle choices—such as beer consumption over milk—and their environment during the war.
Future Research and Implications
Richard H. Davis is eager to submit his findings to medical journals for peer review, hoping to open the door to further research into the environmental and dietary factors that may have contributed to the spread and severity of the Spanish Flu. Davis also draws parallels to the ongoing COVID-19 pandemic, where certain populations seem to have experienced more severe outcomes than others.
While Davis’s milk hypothesis has sparked debate, it offers a unique lens through which to view pandemic preparedness and prevention. "Milk has always been seen as a nutritional powerhouse, but it may also hold the key to protecting future generations from pandemics," said Davis.
https://www.youtube.com/watch?v=L6jTMacxzkI
About Richard H. Davis
Richard H. Davis is an independent researcher specializing in historical and scientific analysis. His work focuses on uncovering hidden connections between environmental factors and global pandemics.
For more information on Richard H. Davis’s paper or to schedule an interview, please call (310) 461-8348.
Category : Environment
Tags : Research
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